APOB

Apolipoprotein B P04114 APOB_HUMAN
Protein Coding Chr 2 2p24.1 Swiss-Prot reviewed Entrez 338
Mutations
4,451
CL 772 · Tissue 3,630
Samples
2,873
CL 512 · Tissue 2,324
Peptides
2,535
unique mutant peptides
Transcripts
3
isoforms mutated

Stats by Source

Total, split by cell line (COSMIC CL / DepMap / PubMed) vs tissue (COSMIC primary tissue)

How the counts split by source
Stats by Source

Total = all mutations for this gene across every source.

Cell line = COSMIC Cell Lines Project + DepMap + PubMed.

Tissue = COSMIC primary-tissue (patient tumour) samples.

Total can exceed cell line + tissue: COSMIC tissue-derived models sit only in global, and a peptide can be shared across both.

TotalCell lineTissue
Mutations4,4517723,630
Samples2,8735122,324
Peptides2,5354062,201

Function

APOB · Apolipoprotein B

This gene product is the main apolipoprotein of chylomicrons and low density lipoproteins (LDL), and is the ligand for the LDL receptor. It occurs in plasma as two main isoforms, apoB-48 and apoB-100: the former is synthesized exclusively in the gut and the latter in the liver. The intestinal and the hepatic forms of apoB are encoded by a single gene from a single, very long mRNA. The two isoforms share a common N-terminal sequence. The shorter apoB-48 protein is produced after RNA editing of the apoB-100 transcript at residue 2180 (CAA->UAA), resulting in the creation of a stop codon, and early translation termination. Mutations in this gene or its regulatory region cause hypobetalipoproteinemia, normotriglyceridemic hypobetalipoproteinemia, and hypercholesterolemia due to ligand-defective apoB, diseases affecting plasma cholesterol and apoB levels. [provided by RefSeq, Dec 2019].

Isoforms & Proteins

3 transcripts · UniProt mapping is sequence-verified (AA-safe)

About the isoform mapping
Isoforms & Proteins

Each Ensembl transcript (ENST) this gene is mutated on, with its matched UniProt accession.

The mapping is sequence-verified: the UniProt sequence is identical to the transcript translation, so amino-acid positions line up exactly. A * marks an unreviewed (TrEMBL) entry.

Counts are mutations and unique mutant peptides on each transcript.

TranscriptUniProtMutationsPeptides
ENST00000233242 P04114 3,904 2,517
ENST00000399256 A8MUN2* 538 383
ENST00000707961 A0AAG2UUW0* 9 9

Gene Properties

Type
Protein Coding
Chromosome
2
Cytoband
2p24.1
Entrez ID
Aliases
FCHL2FLDBLDLCQ4apoB-100apoB-48

Recurrent Mutations

All 2500 amino-acid changes on canonical ENST00000233242 · needle height = samples · drag the mini-map to zoom

What this lollipop shows
Recurrent Mutations

A lollipop / needle plot – the standard way to show recurrent mutations along a protein (as used by cBioPortal and MutationMapper).

X-axis = amino-acid position in the protein.

Needle height & head size = how often that exact amino-acid change was observed (its recurrence). Tall/large heads are mutational hotspots.

The most recurrent changes are labelled; hover any needle for the change, position and counts.

Mutation frequency across cancer types

% of samples with a missense/complex mutation in APOB · cell line vs tissue

How this frequency is counted
Cancer-type mutation frequency

For each cancer type, the fraction of samples that carry at least one missense/complex mutation anywhere in APOB – counted as distinct samples (a sample counts once no matter how many mutations it has).

Split into cell line and tissue; each cell shows mutated / total and the percentage. Cohorts with <20 samples are omitted. Ordered by combined frequency.

Cancer typeCell linesTissue samples
Melanoma
58/210 28%
390/1899 21%
T-Lymphoblastic Leukemia
8/40 20%
0/0 0%
Non-Small Cell Lung Carcinoma
102/304 34%
180/1390 13%
Acute Myeloid Leukemia
14/90 16%
0/0 0%
Oral Cavity Carcinoma
8/54 15%
0/0 0%
Endometrial Carcinoma
10/42 24%
73/612 12%
Squamous Cell Lung Carcinoma
18/57 32%
92/810 11%
Other Solid Cancers
10/94 11%
191/1515 13%
T-Cell Non-Hodgkins Lymphoma
3/26 12%
0/0 0%
Glioblastoma
10/98 10%
0/0 0%
Gastric Carcinoma
7/74 9%
158/1809 9%
Colorectal Carcinoma
47/143 33%
237/3239 7%
Neuroendocrine Tumour
25/154 16%
35/577 6%
Hepatocellular Carcinoma
5/46 11%
137/2210 6%
Bladder Carcinoma
14/58 24%
49/956 5%
Esophageal Carcinoma
0/23 0%
48/769 6%
Ovarian Carcinoma
25/109 23%
40/998 4%
Esophageal Squamous Cell Carcinoma
13/51 25%
137/2550 5%
Other Sarcomas
9/69 13%
35/699 5%
Gastrointestinal Stromal Tumour
0/0 0%
7/133 5%
Osteosarcoma
6/45 13%
5/166 3%
Small Cell Lung Carcinoma
2/9 22%
37/752 5%
Head and Neck Carcinoma
6/85 7%
68/1574 4%
Hodgkins Lymphoma
0/16 0%
6/122 5%
Biliary Tract Carcinoma
7/54 13%
36/950 4%
Cervical Carcinoma
0/35 0%
19/422 4%
Chronic Myelogenous Leukemia
1/25 4%
0/0 0%
Rhabdomyosarcoma
1/33 3%
7/171 4%
Mesothelioma
6/62 10%
1/165 1%
Pancreatic Carcinoma
10/89 11%
40/1611 2%

Mutation Distribution

Where APOB is mutated · all tissues, split by cell line vs tissue

Mutation counts by tissue
Mutation Distribution

How many mutations in APOB were found in each tissue, across the whole database.

Each bar is a tissue (cell-line and tissue names are merged to the standard tissue), split into cell line and tissue (patient tumour) contributions.

This shows the cancer-context where this gene is recurrently altered.

GTEx Expression

Median TPM across 54 healthy tissues

GTEx Portal ↗
About the expression data
GTEx Expression

Median gene expression (TPM) in normal, non-cancer human tissues from the GTEx project.

Useful for judging tumour specificity – a strong neoantigen target ideally comes from a gene with low expression in healthy tissues.

Scroll or drag the mini-axis below the chart to browse all tissues.

Mutations

All 4,451 mutations in APOB

About the mutation list
Mutations

Every mutation record for this gene, across all samples and sources.

The Sample column links to the cell line (cell-line samples) or the tissue type (tissue samples).

Use the Type / Source filters, the search box, and column sorting to explore; each CAN-IMMUNE ID opens the full mutation & peptide view.

IDSampleTranscriptAA Change CDSTypeSourceMutant PeptideWild-type Peptide