CLDN3

Claudin 3 O15551 CLD3_HUMAN
Protein Coding Chr 7 7q11.23 Swiss-Prot reviewed Entrez 1365
Mutations
112
CL 31 · Tissue 80
Samples
102
CL 29 · Tissue 72
Peptides
83
unique mutant peptides
Transcripts
1
isoforms mutated

Stats by Source

Total, split by cell line (COSMIC CL / DepMap / PubMed) vs tissue (COSMIC primary tissue)

How the counts split by source
Stats by Source

Total = all mutations for this gene across every source.

Cell line = COSMIC Cell Lines Project + DepMap + PubMed.

Tissue = COSMIC primary-tissue (patient tumour) samples.

Total can exceed cell line + tissue: COSMIC tissue-derived models sit only in global, and a peptide can be shared across both.

TotalCell lineTissue
Mutations1123180
Samples1022972
Peptides831766

Function

CLDN3 · Claudin 3

Tight junctions represent one mode of cell-to-cell adhesion in epithelial or endothelial cell sheets, forming continuous seals around cells and serving as a physical barrier to prevent solutes and water from passing freely through the paracellular space. These junctions are comprised of sets of continuous networking strands in the outwardly facing cytoplasmic leaflet, with complementary grooves in the inwardly facing extracytoplasmic leaflet. The protein encoded by this intronless gene, a member of the claudin family, is an integral membrane protein and a component of tight junction strands. It is also a low-affinity receptor for Clostridium perfringens enterotoxin, and shares aa sequence similarity with a putative apoptosis-related protein found in rat. [provided by RefSeq, Jul 2008].

Isoforms & Proteins

1 transcript · UniProt mapping is sequence-verified (AA-safe)

About the isoform mapping
Isoforms & Proteins

Each Ensembl transcript (ENST) this gene is mutated on, with its matched UniProt accession.

The mapping is sequence-verified: the UniProt sequence is identical to the transcript translation, so amino-acid positions line up exactly. A * marks an unreviewed (TrEMBL) entry.

Counts are mutations and unique mutant peptides on each transcript.

TranscriptUniProtMutationsPeptides
ENST00000395145 O15551 112 83

Gene Properties

Type
Protein Coding
Chromosome
7
Cytoband
7q11.23
Entrez ID
Aliases
C7orf1CPE-R2CPETR2HRVP1RVP1

Recurrent Mutations

All 83 amino-acid changes on canonical ENST00000395145 · needle height = samples · drag the mini-map to zoom

What this lollipop shows
Recurrent Mutations

A lollipop / needle plot – the standard way to show recurrent mutations along a protein (as used by cBioPortal and MutationMapper).

X-axis = amino-acid position in the protein.

Needle height & head size = how often that exact amino-acid change was observed (its recurrence). Tall/large heads are mutational hotspots.

The most recurrent changes are labelled; hover any needle for the change, position and counts.

Mutation frequency across cancer types

% of samples with a missense/complex mutation in CLDN3 · cell line vs tissue

How this frequency is counted
Cancer-type mutation frequency

For each cancer type, the fraction of samples that carry at least one missense/complex mutation anywhere in CLDN3 – counted as distinct samples (a sample counts once no matter how many mutations it has).

Split into cell line and tissue; each cell shows mutated / total and the percentage. Cohorts with <20 samples are omitted. Ordered by combined frequency.

Cancer typeCell linesTissue samples
T-Lymphoblastic Leukemia
4/40 10%
0/0 0%
Neuroendocrine Tumour
4/154 3%
2/577 0%
Thyroid Gland Carcinoma
1/45 2%
12/1592 1%
Melanoma
4/210 2%
8/1899 0%
Endometrial Carcinoma
2/42 5%
1/612 0%
Cervical Carcinoma
2/35 6%
0/422 0%
Non-Small Cell Lung Carcinoma
3/304 1%
4/1390 0%
Squamous Cell Lung Carcinoma
0/57 0%
3/810 0%
Gastric Carcinoma
1/74 1%
5/1809 0%
B-Cell Non-Hodgkins Lymphoma
0/88 0%
8/2534 0%
Other Solid Cancers
0/94 0%
5/1515 0%
Plasma Cell Myeloma
0/44 0%
1/305 0%
Other Sarcomas
0/69 0%
2/699 0%
Esophageal Carcinoma
0/23 0%
2/769 0%
Colorectal Carcinoma
2/143 1%
6/3239 0%
Glioma
0/52 0%
3/2127 0%
Hepatocellular Carcinoma
0/46 0%
3/2210 0%
Small Cell Lung Carcinoma
0/9 0%
1/752 0%
Non-Cancerous
0/104 0%
1/830 0%
Kidney Carcinoma
1/85 1%
1/1862 0%
Biliary Tract Carcinoma
0/54 0%
1/950 0%
Bladder Carcinoma
0/58 0%
1/956 0%
Prostate Carcinoma
2/13 15%
0/2105 0%
Ovarian Carcinoma
0/109 0%
1/998 0%
B-Lymphoblastic Leukemia
2/55 4%
0/2640 0%
Head and Neck Carcinoma
0/85 0%
1/1574 0%
Esophageal Squamous Cell Carcinoma
0/51 0%
1/2550 0%
Breast Carcinoma
1/144 1%
0/3264 0%

Mutation Distribution

Where CLDN3 is mutated · all tissues, split by cell line vs tissue

Mutation counts by tissue
Mutation Distribution

How many mutations in CLDN3 were found in each tissue, across the whole database.

Each bar is a tissue (cell-line and tissue names are merged to the standard tissue), split into cell line and tissue (patient tumour) contributions.

This shows the cancer-context where this gene is recurrently altered.

GTEx Expression

Median TPM across 54 healthy tissues

GTEx Portal ↗
About the expression data
GTEx Expression

Median gene expression (TPM) in normal, non-cancer human tissues from the GTEx project.

Useful for judging tumour specificity – a strong neoantigen target ideally comes from a gene with low expression in healthy tissues.

Scroll or drag the mini-axis below the chart to browse all tissues.

Mutations

All 112 mutations in CLDN3

About the mutation list
Mutations

Every mutation record for this gene, across all samples and sources.

The Sample column links to the cell line (cell-line samples) or the tissue type (tissue samples).

Use the Type / Source filters, the search box, and column sorting to explore; each CAN-IMMUNE ID opens the full mutation & peptide view.

IDSampleTranscriptAA Change CDSTypeSourceMutant PeptideWild-type Peptide