PRKG1

Protein kinase cGMP-dependent 1 Q13976 KGP1_HUMAN
Protein Coding Chr 10 10q11.23-q21.1 Swiss-Prot reviewed Entrez 5592
Mutations
1,875
CL 250 · Tissue 1,615
Samples
544
CL 101 · Tissue 439
Peptides
487
unique mutant peptides
Transcripts
6
isoforms mutated

Stats by Source

Total, split by cell line (COSMIC CL / DepMap / PubMed) vs tissue (COSMIC primary tissue)

How the counts split by source
Stats by Source

Total = all mutations for this gene across every source.

Cell line = COSMIC Cell Lines Project + DepMap + PubMed.

Tissue = COSMIC primary-tissue (patient tumour) samples.

Total can exceed cell line + tissue: COSMIC tissue-derived models sit only in global, and a peptide can be shared across both.

TotalCell lineTissue
Mutations1,8752501,615
Samples544101439
Peptides48769443

Function

PRKG1 · Protein kinase cGMP-dependent 1

Mammals have three different isoforms of cyclic GMP-dependent protein kinase (Ialpha, Ibeta, and II). These PRKG isoforms act as key mediators of the nitric oxide/cGMP signaling pathway and are important components of many signal transduction processes in diverse cell types. This PRKG1 gene on human chromosome 10 encodes the soluble Ialpha and Ibeta isoforms of PRKG by alternative transcript splicing. A separate gene on human chromosome 4, PRKG2, encodes the membrane-bound PRKG isoform II. The PRKG1 proteins play a central role in regulating cardiovascular and neuronal functions in addition to relaxing smooth muscle tone, preventing platelet aggregation, and modulating cell growth. This gene is most strongly expressed in all types of smooth muscle, platelets, cerebellar Purkinje cells, hippocampal neurons, and the lateral amygdala. Isoforms Ialpha and Ibeta have identical cGMP-binding and catalytic domains but differ in their leucine/isoleucine zipper and autoinhibitory sequences and therefore differ in their dimerization substrates and kinase enzyme activity. [provided by RefSeq, Sep 2011].

Isoforms & Proteins

6 transcripts · UniProt mapping is sequence-verified (AA-safe)

About the isoform mapping
Isoforms & Proteins

Each Ensembl transcript (ENST) this gene is mutated on, with its matched UniProt accession.

The mapping is sequence-verified: the UniProt sequence is identical to the transcript translation, so amino-acid positions line up exactly. A * marks an unreviewed (TrEMBL) entry.

Counts are mutations and unique mutant peptides on each transcript.

TranscriptUniProtMutationsPeptides
ENST00000373980 Q13976-2 588 424
ENST00000401604 Q13976 487 380
ENST00000645324 A0A2R8Y507* 226 179
ENST00000373976 B1ALS0* 224 177
ENST00000643582 A0A2R8YE50* 175 143
ENST00000643704 A0A2R8YH74* 175 143

Gene Properties

Type
Protein Coding
Chromosome
10
Cytoband
10q11.23-q21.1
Entrez ID
Aliases
AAT8PKGPKG1PRKG1BPRKGR1BcGK

Recurrent Mutations

All 424 amino-acid changes on canonical ENST00000373980 · needle height = samples · drag the mini-map to zoom

What this lollipop shows
Recurrent Mutations

A lollipop / needle plot – the standard way to show recurrent mutations along a protein (as used by cBioPortal and MutationMapper).

X-axis = amino-acid position in the protein.

Needle height & head size = how often that exact amino-acid change was observed (its recurrence). Tall/large heads are mutational hotspots.

The most recurrent changes are labelled; hover any needle for the change, position and counts.

Mutation frequency across cancer types

% of samples with a missense/complex mutation in PRKG1 · cell line vs tissue

How this frequency is counted
Cancer-type mutation frequency

For each cancer type, the fraction of samples that carry at least one missense/complex mutation anywhere in PRKG1 – counted as distinct samples (a sample counts once no matter how many mutations it has).

Split into cell line and tissue; each cell shows mutated / total and the percentage. Cohorts with <20 samples are omitted. Ordered by combined frequency.

Cancer typeCell linesTissue samples
T-Cell Non-Hodgkins Lymphoma
5/26 19%
0/0 0%
T-Lymphoblastic Leukemia
4/40 10%
0/0 0%
Endometrial Carcinoma
8/42 19%
19/612 3%
Chronic Myelogenous Leukemia
1/25 4%
0/0 0%
Melanoma
7/210 3%
68/1899 4%
Acute Myeloid Leukemia
3/90 3%
0/0 0%
Squamous Cell Lung Carcinoma
4/57 7%
24/810 3%
Hodgkins Lymphoma
2/16 12%
2/122 2%
Non-Small Cell Lung Carcinoma
11/304 4%
33/1390 2%
Other Solid Cancers
1/94 1%
38/1515 3%
Gastric Carcinoma
0/74 0%
35/1809 2%
Colorectal Carcinoma
16/143 11%
39/3239 1%
Small Cell Lung Carcinoma
2/9 22%
9/752 1%
Bladder Carcinoma
1/58 2%
13/956 1%
Ovarian Carcinoma
6/109 6%
7/998 1%
Head and Neck Carcinoma
3/85 4%
16/1574 1%
Hepatocellular Carcinoma
0/46 0%
26/2210 1%
Neuroendocrine Tumour
5/154 3%
2/577 0%
Biliary Tract Carcinoma
1/54 2%
8/950 1%
Burkitts Lymphoma
2/32 6%
0/196 0%
Other Sarcomas
2/69 3%
4/699 1%
Esophageal Carcinoma
1/23 4%
5/769 1%
Esophageal Squamous Cell Carcinoma
1/51 2%
18/2550 1%
Breast Carcinoma
5/144 3%
19/3264 1%
B-Cell Non-Hodgkins Lymphoma
5/88 6%
13/2534 1%
Plasma Cell Myeloma
0/44 0%
2/305 1%
Thyroid Gland Carcinoma
0/45 0%
9/1592 1%
Osteosarcoma
1/45 2%
0/166 0%
Mesothelioma
1/62 2%
0/165 0%
Cervical Carcinoma
0/35 0%
2/422 0%

Mutation Distribution

Where PRKG1 is mutated · all tissues, split by cell line vs tissue

Mutation counts by tissue
Mutation Distribution

How many mutations in PRKG1 were found in each tissue, across the whole database.

Each bar is a tissue (cell-line and tissue names are merged to the standard tissue), split into cell line and tissue (patient tumour) contributions.

This shows the cancer-context where this gene is recurrently altered.

GTEx Expression

Median TPM across 54 healthy tissues

GTEx Portal ↗
About the expression data
GTEx Expression

Median gene expression (TPM) in normal, non-cancer human tissues from the GTEx project.

Useful for judging tumour specificity – a strong neoantigen target ideally comes from a gene with low expression in healthy tissues.

Scroll or drag the mini-axis below the chart to browse all tissues.

Mutations

All 1,875 mutations in PRKG1

About the mutation list
Mutations

Every mutation record for this gene, across all samples and sources.

The Sample column links to the cell line (cell-line samples) or the tissue type (tissue samples).

Use the Type / Source filters, the search box, and column sorting to explore; each CAN-IMMUNE ID opens the full mutation & peptide view.

IDSampleTranscriptAA Change CDSTypeSourceMutant PeptideWild-type Peptide