RP1

RP1 axonemal microtubule associated P56715 RP1_HUMAN
Protein Coding Chr 8 8q11.23-q12.1 Swiss-Prot reviewed Entrez 6101
Mutations
3,839
CL 515 · Tissue 3,299
Samples
2,123
CL 361 · Tissue 1,745
Peptides
1,711
unique mutant peptides
Transcripts
3
isoforms mutated

Stats by Source

Total, split by cell line (COSMIC CL / DepMap / PubMed) vs tissue (COSMIC primary tissue)

How the counts split by source
Stats by Source

Total = all mutations for this gene across every source.

Cell line = COSMIC Cell Lines Project + DepMap + PubMed.

Tissue = COSMIC primary-tissue (patient tumour) samples.

Total can exceed cell line + tissue: COSMIC tissue-derived models sit only in global, and a peptide can be shared across both.

TotalCell lineTissue
Mutations3,8395153,299
Samples2,1233611,745
Peptides1,7112931,480

Function

RP1 · RP1 axonemal microtubule associated

This gene encodes a member of the doublecortin family. The protein encoded by this gene contains two doublecortin domains, which bind microtubules and regulate microtubule polymerization. The encoded protein is a photoreceptor microtubule-associated protein and is required for correct stacking of outer segment disc. This protein and the RP1L1 protein, another retinal-specific protein, play essential and synergistic roles in affecting photosensitivity and outer segment morphogenesis of rod photoreceptors. Because of its response to in vivo retinal oxygen levels, this protein was initially named ORP1 (oxygen-regulated protein-1). This protein was subsequently designated RP1 (retinitis pigmentosa 1) when it was found that mutations in this gene cause autosomal dominant retinitis pigmentosa. Mutations in this gene also cause autosomal recessive retinitis pigmentosa. Transcript variants resulted from an alternative promoter and alternative splicings have been found, which overlap the current reference sequence and has several exons upstream and downstream of the current reference sequence. However, the biological validity and full-length nature of some variants cannot be determined at this time.[provided by RefSeq, Sep 2010].

Isoforms & Proteins

3 transcripts · UniProt mapping is sequence-verified (AA-safe)

About the isoform mapping
Isoforms & Proteins

Each Ensembl transcript (ENST) this gene is mutated on, with its matched UniProt accession.

The mapping is sequence-verified: the UniProt sequence is identical to the transcript translation, so amino-acid positions line up exactly. A * marks an unreviewed (TrEMBL) entry.

Counts are mutations and unique mutant peptides on each transcript.

TranscriptUniProtMutationsPeptides
ENST00000220676 P56715 2,613 1,506
ENST00000636932 A0A1B0GTV9* 619 397
ENST00000637698 A0A1B0GUH0* 607 384

Gene Properties

Type
Protein Coding
Chromosome
8
Cytoband
8q11.23-q12.1
Entrez ID
Aliases
DCDC4AORP1

Recurrent Mutations

All 1506 amino-acid changes on canonical ENST00000220676 · needle height = samples · drag the mini-map to zoom

What this lollipop shows
Recurrent Mutations

A lollipop / needle plot – the standard way to show recurrent mutations along a protein (as used by cBioPortal and MutationMapper).

X-axis = amino-acid position in the protein.

Needle height & head size = how often that exact amino-acid change was observed (its recurrence). Tall/large heads are mutational hotspots.

The most recurrent changes are labelled; hover any needle for the change, position and counts.

Mutation frequency across cancer types

% of samples with a missense/complex mutation in RP1 · cell line vs tissue

How this frequency is counted
Cancer-type mutation frequency

For each cancer type, the fraction of samples that carry at least one missense/complex mutation anywhere in RP1 – counted as distinct samples (a sample counts once no matter how many mutations it has).

Split into cell line and tissue; each cell shows mutated / total and the percentage. Cohorts with <20 samples are omitted. Ordered by combined frequency.

Cancer typeCell linesTissue samples
Melanoma
63/210 30%
432/1899 23%
T-Lymphoblastic Leukemia
6/40 15%
0/0 0%
Endometrial Carcinoma
14/42 33%
47/612 8%
Other Solid Cancers
7/94 7%
136/1515 9%
Squamous Cell Lung Carcinoma
9/57 16%
62/810 8%
Non-Small Cell Lung Carcinoma
41/304 13%
92/1390 7%
Gastric Carcinoma
6/74 8%
132/1809 7%
Glioblastoma
6/98 6%
0/0 0%
Colorectal Carcinoma
41/143 29%
155/3239 5%
Esophageal Squamous Cell Carcinoma
16/51 31%
121/2550 5%
Esophageal Carcinoma
0/23 0%
41/769 5%
Unknown
0/10 0%
2/29 7%
Gastrointestinal Stromal Tumour
0/0 0%
6/133 5%
Acute Myeloid Leukemia
4/90 4%
0/0 0%
Rhabdomyosarcoma
0/33 0%
9/171 5%
Chronic Myelogenous Leukemia
1/25 4%
0/0 0%
Small Cell Lung Carcinoma
0/9 0%
30/752 4%
T-Cell Non-Hodgkins Lymphoma
1/26 4%
0/0 0%
Oral Cavity Carcinoma
2/54 4%
0/0 0%
Hodgkins Lymphoma
4/16 25%
1/122 1%
Hepatocellular Carcinoma
10/46 22%
69/2210 3%
Neuroendocrine Tumour
19/154 12%
6/577 1%
Other Sarcomas
7/69 10%
18/699 3%
Head and Neck Carcinoma
6/85 7%
45/1574 3%
Plasma Cell Myeloma
7/44 16%
3/305 1%
Bladder Carcinoma
1/58 2%
28/956 3%
Burkitts Lymphoma
6/32 19%
0/196 0%
Cervical Carcinoma
4/35 11%
8/422 2%
Germ Cell Tumour
3/25 12%
2/169 1%
Ovarian Carcinoma
7/109 6%
17/998 2%

Mutation Distribution

Where RP1 is mutated · all tissues, split by cell line vs tissue

Mutation counts by tissue
Mutation Distribution

How many mutations in RP1 were found in each tissue, across the whole database.

Each bar is a tissue (cell-line and tissue names are merged to the standard tissue), split into cell line and tissue (patient tumour) contributions.

This shows the cancer-context where this gene is recurrently altered.

GTEx Expression

Median TPM across 50 healthy tissues

GTEx Portal ↗
About the expression data
GTEx Expression

Median gene expression (TPM) in normal, non-cancer human tissues from the GTEx project.

Useful for judging tumour specificity – a strong neoantigen target ideally comes from a gene with low expression in healthy tissues.

Scroll or drag the mini-axis below the chart to browse all tissues.

Mutations

All 3,839 mutations in RP1

About the mutation list
Mutations

Every mutation record for this gene, across all samples and sources.

The Sample column links to the cell line (cell-line samples) or the tissue type (tissue samples).

Use the Type / Source filters, the search box, and column sorting to explore; each CAN-IMMUNE ID opens the full mutation & peptide view.

IDSampleTranscriptAA Change CDSTypeSourceMutant PeptideWild-type Peptide